ORAI Channels as Potential Therapeutic Targets in Pulmonary Hypertension.

Publié en July 2018 Physiology (Bethesda, Md.)

NiONPs-induced alteration in calcium signaling and mitochondrial function in pulmonary artery endothelial cells involves oxidative stress and TRPV4 channels disruption.

Physiology (Bethesda, Md.)

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In vitro study of carbon black nanoparticles on human pulmonary artery endothelial cells: effects on calcium signaling and mitochondrial alterations.

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