When the close relationship between function and substrate metabolism in ex vivo working mouse hearts goes beyond differences in genetic background

Publié en January 2012 FASEB JOURNAL

Ivabradine but not propranolol delays the time to onset of ischaemia-induced ventricular fibrillation by preserving myocardial metabolic energy status

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Heart rate reduction with ivabradine increases ischaemia-induced ventricular fibrillation threshold: Role of myocyte structure and myocardial perfusion

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