Elise MAURAT

Ingénieure d'étude

Parcours professionnel

Depuis 2012 : Ingénieure d’étude en biologie cellulaire – CRCTB, INSERM U1045, Université de Bordeaux

2008-2011 : Technicienne de recherche – MRGM, INSERM U1211, Université de Bordeaux
Recherche de mutations génétiques et études des conséquences fonctionnelles dans les maladies neurodégénératives (ARSACS, Hétérotopies Nodulaires périventriculaires)

2006-2007 : Technicienne en recherche et développement – MDS Pharma Service, Lyon(69)
Pharmacovigilances de molécules à visées thérapeutiques

2005 : Technicienne en recherche et développement – Servier, Orleans(45)
Screening de molécules à visées thérapeutiques

2004-2005 : Technicienne en recherche et développement – CERB, Bourges(18)
Pharmacovigilances de molécules à visées thérapeutiques

Formation
2004 : DUT ABB – Université de Clermont-Auvergne
2017 : Licence GBMQ – Université de Nice
2025 : Diplôme d’établissement SVT – EPHE, Université Paris Sciences et Lettres

Membres

Elise MAURAT

Parcours professionnel

Publications 41

CXCR4 Blockade Alleviates Pulmonary and Cardiac Outcomes in Early Chronic Obstructive Pulmonary Disease

Tunable 3D Alveolosphere Model from Human Alveolar Cells: A Breakthrough Tool to Explore Emphysema Pathophysiology

LSC-2025-Modeling COPD with a complex tubular distal airway model integrating the epithelial and mesenchymal compartments

A novel in vitro tubular model to recapitulate features of distal airways: The bronchioid

Exploring effects of mechanical stress in the development of emphysema using a lung organoid

Targeting mucus hyperproduction in COPD using siRNA-loaded switchable lipid nanoparticles to silence SPDEF

Involvement of the CXCR4/CXCL12 axis in skeletal muscle wasting in a murine model of Chronic Obstructive Pulmonary Disease (COPD)

Blocking CXCR4 improves pulmonary and cardiac outcomes in a mouse model of early COPD

Probabilistic cellular automata modelling of intercellular interactions in airways: complex pattern formation in patients with chronic obstructive pulmonary disease

CXCR4 blockade alleviates pulmonary and cardiac outcomes in young COPD

Rhinovirus infection of bronchial epithelium induces specific bronchial smooth muscle cell migration of severe asthmatic patients

Asthmatic bronchial smooth muscle increases rhinovirus replication within the bronchial epithelium

Muscarinic receptor M3 activation promotes fibrocytes contraction

A high probability of short-range interactions between fibrocytes and CD8+T cells potentiates the inflammatory response in COPD

Asthmatic Bronchial Smooth Muscle Decreases Epithelial Response to Rhinovirus Infection Through a CCL20-EIF2AK2 Dependent Pathway

Circulating fibrocytes as a new tool to predict lung cancer progression after surgery?

Short-range interactions between fibrocytes and CD8+T cells modulate the balance between tissue repair and destruction in COPD

Crucial role of fatty acid oxidation in asthmatic bronchial smooth muscle remodeling

Bronchial smooth muscle of severe asthmatic patients decreases rhinovirus replication within the epithelium through a CCL-20/PKR/eIF2-a dependent pathway

Human rhinovirus induce specific bronchial smooth muscle migration toward bronchial epithelium in severe asthma through the CXCL10/CXCR3 axis

Crucial role of fatty acid oxidation in asthmatic bronchial smooth muscle remodelling

The burden of emphysema on lung cancer prognosis

A high probability of short-range interactions between fibrocytes and CD8+T cells potentiates the inflammatory response in chronic obstructive pulmonary disease (COPD) lungs

Bronchial Smooth Muscle of Severe Asthmatic Patients Decreases Rhinovirus Replication Within the Epithelium Through a CCL-20/PkR/eIF2alpha-Dependent Pathway

Implication of Mitochondria! Metabolism in Bronchial Smooth Muscle Remodeling in Asthma

Connexin-43 is a promising target for pulmonary hypertension due to hypoxaemic lung disease

Asthmatic Bronchial Smooth Muscle Increases CCL5-Dependent Monocyte Migration in Response to Rhinovirus-Infected Epithelium

Fibrocyte accumulation in the airway walls of COPD patients

A high probability of short-range interactions between fibrocytes and CD8 T cells in chronic obstructive pulmonary disease (COPD) lungs

Fibrocyte accumulation in bronchi: a cellular hallmark of COPD

Bronchial fibrocyte accumulation in COPD patients

Blood fibrocytes are recruited during acute exacerbations of chronic obstructive pulmonary disease through a CXCR4-dependent pathway

Selective dysfunction of p53 for mitochondrial biogenesis induces cellular proliferation in bronchial smooth muscle from asthmatic patients

New practical definitions for the diagnosis of autosomal recessive spastic ataxia of Charlevoix-Saguenay

Increased Bronchial Smooth Muscle Mass And Mitochondrial Biogenesis In Non-Severe Asthma

The critical role of bronchial smooth muscle remodeling in non-severe asthma

P53 dysfunction increased mitochondrial biogenesis and bronchial smooth muscle cell proliferation in asthma

Recruitment of blood fibrocytes during acute exacerbations of chronic obstructive pulmonary disease through a CXCR4 dependent pathway

Protease activated receptor-2 expression and function in asthmatic bronchial smooth muscle

The role of fibrocytes in bronchial remodelling during acute exacerbation of COPD

Atypical male and female presentations of FLNA-related periventricular nodular heterotopia

Équipe

Remodelage bronchique